All EMS presentations
RespiratoryClinical presentation

Shortness of breath - undifferentiated

A streamlined, differential-led presentation for rapid clinical review.

Clinical snapshot

Twenty-second review

  • Dyspnea is the perception of respiratory effort produced by altered mechanics, gas exchange, circulatory function, metabolic demand, or central drive.
  • Silent chest, exhaustion, inability to speak, cyanosis, shock, unilateral absent sounds, or decreasing LOC signals impending respiratory failure.
  • Keep metabolic acidosis or anxiety in the differential; overlapping or concurrent illness remains possible.
  • For shortness of breath - undifferentiated, trends and combinations of findings are more informative than one isolated vital sign.
Differentials

Compare the clinical patterns

The repeated material is grouped inside each possibility. Open only the condition you need.

Serious2 conditions
Pulmonary embolismTime-sensitive high-risk possibility

HPI

Clarify sudden or gradual; degree of distress; spo2 on room air and now.

Signs & symptoms

Sudden distress, pleuritic pain, shock, or asymmetric findings.

Focused exam

Observe position, speech, accessory-muscle use, pattern, depth, and chest symmetry; Auscultate all lung fields and reassess after movement or treatment.

Brief pathophysiology

Pulmonary embolism may cause shortness of breath - undifferentiated through a time-sensitive process capable of rapid physiological deterioration.

Vital-sign pattern

What the trend may show

Possible pattern

Pulmonary embolism may have normal observations. Tachycardia, tachypnea, or hypoxemia increase concern; hypotension identifies hemodynamic compromise rather than being required for diagnosis.

Heart rate (bpm)

May rise

Tachycardia is common but neither sensitive nor specific.

Respiratory rate (breaths/min)

May rise

Tachypnea may be an early sign of increased ventilatory demand.

Oxygen saturation (SpO₂)

May fall

Hypoxemia may occur, but a normal saturation does not exclude PE.

Blood pressure (mmHg)

Often preserved

Hypotension suggests high-risk PE with obstructive shock.

Front-load in the triage report

  • Pulmonary embolism: Sudden distress, pleuritic pain, shock, or asymmetric findings.
  • Sudden or gradual?
  • Degree of distress.
Tension pneumothoraxTime-sensitive high-risk possibility

HPI

Clarify degree of distress; spo2 on room air and now; new oxygen requirement.

Signs & symptoms

Sudden distress, pleuritic pain, shock, or asymmetric findings.

Focused exam

Auscultate all lung fields and reassess after movement or treatment; Compare air entry and sounds bilaterally; inspect effort, position, edema, JVP, chest pain, and DVT signs.

Brief pathophysiology

Tension pneumothorax may cause shortness of breath - undifferentiated through a time-sensitive process capable of rapid physiological deterioration.

Vital-sign pattern

What the trend may show

Possible pattern

Thoracic injury can evolve from respiratory compromise to obstructive or hemorrhagic shock. Integrate symmetry, air entry, work of breathing, and perfusion with serial observations.

Respiratory rate (breaths/min)

Often rises

Tachypnea commonly reflects pain, hypoxemia, or impaired mechanics.

Oxygen saturation (SpO₂)

May fall

Desaturation supports significant gas-exchange impairment but may lag.

Heart rate (bpm)

Often rises

Tachycardia may accompany distress, hypoxia, or shock.

Blood pressure (mmHg)

May fall late

Hypotension raises concern for tension physiology or significant hemorrhage.

Front-load in the triage report

  • Tension pneumothorax: Sudden distress, pleuritic pain, shock, or asymmetric findings.
  • Degree of distress.
  • SpO2 on room air and now.
Common2 conditions
ObstructiveCommon presentation pattern

HPI

Clarify sudden or gradual; degree of distress; spo2 on room air and now.

Signs & symptoms

Wheeze, cough, fever, sputum, or known asthma/COPD may guide the working impression.

Focused exam

Observe position, speech, accessory-muscle use, pattern, depth, and chest symmetry; Auscultate all lung fields and reassess after movement or treatment.

Brief pathophysiology

Obstructive may explain the presenting symptoms through a common clinical mechanism, while serial assessment checks for complications or an alternate cause.

Vital-sign pattern

What the trend may show

Possible pattern

Shock can be present before profound hypotension. Trend perfusion, mental status, pressure, and pulse characteristics; the direction varies with cause and treatment.

Blood pressure (mmHg)

May fall

Relative change from baseline may be important before absolute hypotension appears.

MAP (mmHg)

May fall

A falling MAP supports impaired perfusion but is not the only endpoint.

Heart rate (bpm)

Often rises

Tachycardia is common; neurogenic, toxic, paced, or medication effects can alter the response.

Pulse pressure (mmHg)

May narrow

Narrow pulse pressure can reflect reduced stroke volume, although distributive states may differ.

Front-load in the triage report

  • Wheeze, cough, fever, sputum, or known asthma/COPD may guide the working impression.
  • Sudden or gradual?
  • Degree of distress.
Infectious lung diseaseCommon presentation pattern

HPI

Clarify degree of distress; spo2 on room air and now; new oxygen requirement.

Signs & symptoms

Wheeze, cough, fever, sputum, or known asthma/COPD may guide the working impression.

Focused exam

Auscultate all lung fields and reassess after movement or treatment; Compare air entry and sounds bilaterally; inspect effort, position, edema, JVP, chest pain, and DVT signs.

Brief pathophysiology

Infectious lung disease may explain the presenting symptoms through a common clinical mechanism, while serial assessment checks for complications or an alternate cause.

Vital-sign pattern

What the trend may show

Possible pattern

Respiratory presentations require serial assessment of work of breathing and ventilation as well as oxygenation. A normal SpO₂ does not rule out serious ventilatory disease.

Respiratory rate (breaths/min)

Trend rate / effort

A rising rate suggests distress; a late fall with fatigue may be ominous.

Oxygen saturation (SpO₂)

Trend against baseline

Interpret the number with work of breathing and device limitations.

Heart rate (bpm)

May rise

Rate may reflect hypoxia, distress, treatment, or another cause.

Blood pressure (mmHg)

Trend perfusion

Hypotension suggests advanced disease or a concurrent shock state.

Front-load in the triage report

  • Wheeze, cough, fever, sputum, or known asthma/COPD may guide the working impression.
  • Degree of distress.
  • SpO2 on room air and now.
Mimics2 conditions
Metabolic acidosisImportant alternative presentation

HPI

Clarify sudden or gradual; degree of distress; spo2 on room air and now.

Signs & symptoms

Rapid breathing with relatively clear lungs requires broader assessment.

Focused exam

Observe position, speech, accessory-muscle use, pattern, depth, and chest symmetry; Auscultate all lung fields and reassess after movement or treatment.

Brief pathophysiology

Metabolic acidosis can resemble shortness of breath - undifferentiated through overlapping symptoms or referred physiology despite a different primary mechanism.

Vital-sign pattern

What the trend may show

Possible pattern

Respiratory presentations require serial assessment of work of breathing and ventilation as well as oxygenation. A normal SpO₂ does not rule out serious ventilatory disease.

Respiratory rate (breaths/min)

Trend rate / effort

A rising rate suggests distress; a late fall with fatigue may be ominous.

Oxygen saturation (SpO₂)

Trend against baseline

Interpret the number with work of breathing and device limitations.

Heart rate (bpm)

May rise

Rate may reflect hypoxia, distress, treatment, or another cause.

Blood pressure (mmHg)

Trend perfusion

Hypotension suggests advanced disease or a concurrent shock state.

Front-load in the triage report

  • Rapid breathing with relatively clear lungs requires broader assessment.
  • Sudden or gradual?
  • Degree of distress.
AnxietyImportant alternative presentation

HPI

Clarify degree of distress; spo2 on room air and now; new oxygen requirement.

Signs & symptoms

Rapid breathing with relatively clear lungs requires broader assessment.

Focused exam

Auscultate all lung fields and reassess after movement or treatment; Compare air entry and sounds bilaterally; inspect effort, position, edema, JVP, chest pain, and DVT signs.

Brief pathophysiology

Anxiety can resemble shortness of breath - undifferentiated through overlapping symptoms or referred physiology despite a different primary mechanism.

Vital-sign pattern

What the trend may show

Possible pattern

Respiratory presentations require serial assessment of work of breathing and ventilation as well as oxygenation. A normal SpO₂ does not rule out serious ventilatory disease.

Respiratory rate (breaths/min)

Trend rate / effort

A rising rate suggests distress; a late fall with fatigue may be ominous.

Oxygen saturation (SpO₂)

Trend against baseline

Interpret the number with work of breathing and device limitations.

Heart rate (bpm)

May rise

Rate may reflect hypoxia, distress, treatment, or another cause.

Blood pressure (mmHg)

Trend perfusion

Hypotension suggests advanced disease or a concurrent shock state.

Front-load in the triage report

  • Rapid breathing with relatively clear lungs requires broader assessment.
  • Degree of distress.
  • SpO2 on room air and now.
Red flags

Findings that should escalate concern

  • Silent chest, exhaustion, inability to speak, cyanosis, shock, unilateral absent sounds, or decreasing LOC signals impending respiratory failure.
  • Pulmonary embolism or tension pneumothorax: Sudden distress, pleuritic pain, shock, or asymmetric findings.
Related medications

Medication recognition

Related EMS presentations

Continue the clinical pathway

References

Clinical and academic sources

Curated clinical guidance and peer-reviewed evidence supporting this presentation. Current BCEHS guidance, medical direction, and local policy remain authoritative for practice.

BCEHS Handbook — specific CPGs

  1. A02: Patient AssessmentBC Emergency Health Services Handbook

    Official BCEHS clinical practice guidance for patient assessment.

  2. A07: Oxygen AdministrationBC Emergency Health Services Handbook

    Official BCEHS clinical practice guidance for oxygen administration.

  3. B01: Airway ManagementBC Emergency Health Services Handbook

    Official BCEHS clinical practice guidance for airway management.

Supporting clinical and academic evidence

  1. CEDIS Presenting Complaint List updateCanadian Association of Emergency Physicians

    Presenting-complaint terminology and categorization context. It does not establish a prehospital “top 100” ranking.

  2. Guideline for oxygen use in adults in healthcare and emergency settingsBritish Thoracic Society

    Peer-reviewed guidance for oxygen assessment, targets, delivery, and monitoring in acute illness.

  3. Acute dyspnea in the emergency department: a clinical reviewInternal and Emergency Medicine · 2023

    Differential-led assessment of acute dyspnea, respiratory effort, gas exchange, and time-sensitive causes.