Urinary complaint / UTI / retention
A streamlined, differential-led presentation for rapid clinical review.
Twenty-second review
- Urinary symptoms may reflect infection, obstruction, retention, stone disease, bleeding, medication effect, or neurologic dysfunction and can progress to renal injury or sepsis.
- Anuria, severe retention, fever with shock, flank pain with infection, gross bleeding with clots, or new neurologic deficits requires escalation.
- Keep medication or neurologic cause in the differential; overlapping or concurrent illness remains possible.
- For urinary complaint / uti / retention, trends and combinations of findings are more informative than one isolated vital sign.
Compare the clinical patterns
The repeated material is grouped inside each possibility. Open only the condition you need.
PyelonephritisTime-sensitive high-risk possibility
HPI
Clarify dysuria/frequency; unable to void; last urination.
Signs & symptoms
Fever, flank pain, vomiting, hypotension, or reduced urine output.
Focused exam
Assess hydration, edema, lung sounds, bladder or flank tenderness, urine appearance, and dialysis access when relevant; Obtain a 12-lead ECG when electrolyte disturbance is plausible.
Brief pathophysiology
Pyelonephritis may cause urinary complaint / uti / retention through a time-sensitive process capable of rapid physiological deterioration.
What the trend may show
Genitourinary and renal differentials can involve pain, infection, volume change, or metabolic disturbance. These observations help distinguish local from systemic illness.
Temperature (°C)
Trend temperatureFever or hypothermia raises concern for systemic infection.
Blood pressure (mmHg)
High or lowRenal disease, volume overload, or dehydration can alter pressure in either direction.
Heart rate (bpm)
May risePain, fever, dehydration, and shock can increase rate.
Respiratory rate (breaths/min)
May riseTachypnea may reflect sepsis, acidosis, pain, or pulmonary edema.
Front-load in the triage report
- Pyelonephritis: Fever, flank pain, vomiting, hypotension, or reduced urine output.
- Dysuria/frequency.
- Unable to void?
UrosepsisTime-sensitive high-risk possibility
HPI
Clarify unable to void; last urination; hematuria.
Signs & symptoms
Fever, flank pain, vomiting, hypotension, or reduced urine output.
Focused exam
Obtain a 12-lead ECG when electrolyte disturbance is plausible; Assess bladder distension, suprapubic or flank tenderness, hydration, urine character, catheter function, edema, and infection signs.
Brief pathophysiology
Urosepsis may cause urinary complaint / uti / retention through a time-sensitive process capable of rapid physiological deterioration.
What the trend may show
No single vital sign diagnoses sepsis. New tachypnea, abnormal temperature, tachycardia, or falling pressure—especially with altered mentation or poor perfusion—supports organ dysfunction and urgency.
Respiratory rate (breaths/min)
May rise earlyTachypnea is included in NEWS2 and can precede overt hypotension.
Temperature (°C)
High or lowAbsence of fever does not exclude sepsis; hypothermia is concerning.
Heart rate (bpm)
Often risesTachycardia is common but may be blunted by age or medication.
Blood pressure (mmHg)
May fallRelative hypotension and trend matter; profound hypotension can be late.
Front-load in the triage report
- Urosepsis: Fever, flank pain, vomiting, hypotension, or reduced urine output.
- Unable to void?
- Last urination.
Obstructive renal injuryTime-sensitive high-risk possibility
HPI
Clarify last urination; hematuria; fever/rigors.
Signs & symptoms
Fever, flank pain, vomiting, hypotension, or reduced urine output.
Focused exam
Obtain a 12-lead ECG when electrolyte disturbance is plausible; Assess bladder distension, suprapubic or flank tenderness, hydration, urine character, catheter function, edema, and infection signs.
Brief pathophysiology
Obstructive renal injury may cause urinary complaint / uti / retention through a time-sensitive process capable of rapid physiological deterioration.
What the trend may show
Shock can be present before profound hypotension. Trend perfusion, mental status, pressure, and pulse characteristics; the direction varies with cause and treatment.
Blood pressure (mmHg)
May fallRelative change from baseline may be important before absolute hypotension appears.
MAP (mmHg)
May fallA falling MAP supports impaired perfusion but is not the only endpoint.
Heart rate (bpm)
Often risesTachycardia is common; neurogenic, toxic, paced, or medication effects can alter the response.
Pulse pressure (mmHg)
May narrowNarrow pulse pressure can reflect reduced stroke volume, although distributive states may differ.
Front-load in the triage report
- Obstructive renal injury: Fever, flank pain, vomiting, hypotension, or reduced urine output.
- Last urination.
- Hematuria.
Lower UTICommon presentation pattern
HPI
Clarify dysuria/frequency; unable to void; last urination.
Signs & symptoms
Dysuria, frequency, suprapubic discomfort, poor stream, or palpable bladder may fit.
Focused exam
Assess hydration, edema, lung sounds, bladder or flank tenderness, urine appearance, and dialysis access when relevant; Obtain a 12-lead ECG when electrolyte disturbance is plausible.
Brief pathophysiology
Lower UTI may explain the presenting symptoms through a common clinical mechanism, while serial assessment checks for complications or an alternate cause.
What the trend may show
Genitourinary and renal differentials can involve pain, infection, volume change, or metabolic disturbance. These observations help distinguish local from systemic illness.
Temperature (°C)
Trend temperatureFever or hypothermia raises concern for systemic infection.
Blood pressure (mmHg)
High or lowRenal disease, volume overload, or dehydration can alter pressure in either direction.
Heart rate (bpm)
May risePain, fever, dehydration, and shock can increase rate.
Respiratory rate (breaths/min)
May riseTachypnea may reflect sepsis, acidosis, pain, or pulmonary edema.
Front-load in the triage report
- Dysuria, frequency, suprapubic discomfort, poor stream, or palpable bladder may fit.
- Dysuria/frequency.
- Unable to void?
RetentionCommon presentation pattern
HPI
Clarify unable to void; last urination; hematuria.
Signs & symptoms
Dysuria, frequency, suprapubic discomfort, poor stream, or palpable bladder may fit.
Focused exam
Obtain a 12-lead ECG when electrolyte disturbance is plausible; Assess bladder distension, suprapubic or flank tenderness, hydration, urine character, catheter function, edema, and infection signs.
Brief pathophysiology
Retention may explain the presenting symptoms through a common clinical mechanism, while serial assessment checks for complications or an alternate cause.
What the trend may show
Genitourinary and renal differentials can involve pain, infection, volume change, or metabolic disturbance. These observations help distinguish local from systemic illness.
Temperature (°C)
Trend temperatureFever or hypothermia raises concern for systemic infection.
Blood pressure (mmHg)
High or lowRenal disease, volume overload, or dehydration can alter pressure in either direction.
Heart rate (bpm)
May risePain, fever, dehydration, and shock can increase rate.
Respiratory rate (breaths/min)
May riseTachypnea may reflect sepsis, acidosis, pain, or pulmonary edema.
Front-load in the triage report
- Dysuria, frequency, suprapubic discomfort, poor stream, or palpable bladder may fit.
- Unable to void?
- Last urination.
MedicationImportant alternative presentation
HPI
Clarify dysuria/frequency; unable to void; last urination.
Signs & symptoms
Anticholinergic drugs, prostate disease, or spinal pathology can produce retention.
Focused exam
Assess hydration, edema, lung sounds, bladder or flank tenderness, urine appearance, and dialysis access when relevant; Obtain a 12-lead ECG when electrolyte disturbance is plausible.
Brief pathophysiology
Medication can resemble urinary complaint / uti / retention through overlapping symptoms or referred physiology despite a different primary mechanism.
What the trend may show
Genitourinary and renal differentials can involve pain, infection, volume change, or metabolic disturbance. These observations help distinguish local from systemic illness.
Temperature (°C)
Trend temperatureFever or hypothermia raises concern for systemic infection.
Blood pressure (mmHg)
High or lowRenal disease, volume overload, or dehydration can alter pressure in either direction.
Heart rate (bpm)
May risePain, fever, dehydration, and shock can increase rate.
Respiratory rate (breaths/min)
May riseTachypnea may reflect sepsis, acidosis, pain, or pulmonary edema.
Front-load in the triage report
- Anticholinergic drugs, prostate disease, or spinal pathology can produce retention.
- Dysuria/frequency.
- Unable to void?
Neurologic causeImportant alternative presentation
HPI
Clarify unable to void; last urination; hematuria.
Signs & symptoms
Anticholinergic drugs, prostate disease, or spinal pathology can produce retention.
Focused exam
Obtain a 12-lead ECG when electrolyte disturbance is plausible; Assess bladder distension, suprapubic or flank tenderness, hydration, urine character, catheter function, edema, and infection signs.
Brief pathophysiology
Neurologic cause can resemble urinary complaint / uti / retention through overlapping symptoms or referred physiology despite a different primary mechanism.
What the trend may show
Genitourinary and renal differentials can involve pain, infection, volume change, or metabolic disturbance. These observations help distinguish local from systemic illness.
Temperature (°C)
Trend temperatureFever or hypothermia raises concern for systemic infection.
Blood pressure (mmHg)
High or lowRenal disease, volume overload, or dehydration can alter pressure in either direction.
Heart rate (bpm)
May risePain, fever, dehydration, and shock can increase rate.
Respiratory rate (breaths/min)
May riseTachypnea may reflect sepsis, acidosis, pain, or pulmonary edema.
Front-load in the triage report
- Anticholinergic drugs, prostate disease, or spinal pathology can produce retention.
- Unable to void?
- Last urination.
Findings that should escalate concern
- Anuria, severe retention, fever with shock, flank pain with infection, gross bleeding with clots, or new neurologic deficits requires escalation.
- Pyelonephritis, urosepsis, or obstructive renal injury: Fever, flank pain, vomiting, hypotension, or reduced urine output.
Medication recognition
Continue the clinical pathway
Clinical and academic sources
Curated clinical guidance and peer-reviewed evidence supporting this presentation. Current BCEHS guidance, medical direction, and local policy remain authoritative for practice.
BCEHS Handbook — specific CPGs
Supporting clinical and academic evidence
- CEDIS Presenting Complaint List updateCanadian Association of Emergency Physicians
Presenting-complaint terminology and categorization context. It does not establish a prehospital “top 100” ranking.
- Acute kidney injury: prevention, detection and managementNational Institute for Health and Care Excellence
Clinical guidance for recognizing renal risk, oliguria, obstruction, electrolyte complications, and deterioration.
- Urologic EmergenciesMedical Clinics of North America · 2018
Recognition of urinary obstruction, infection, retention, hematuria, and other time-sensitive urologic conditions.